The bacterial metabolite indole-3-propionic acid (IpRA) increases brain levels of kynurenic acid (KYNA) in an in vivo murine model.

Authors

  • Tonali Blanco Ayala Laboratorio de Neurobioquímica y Conducta, Instituto Naicional de Neurología y Neurocirugía Manuel Velasco Suárez
  • Korrapati Sathyasaikumar Maryland Psychiatric Research Center, University of Maryland
  • Yiran Zheng Dept. of Physiology and Pharmacology, Karolinska Institute
  • Lily Schwieler Dept. of Physiology and Pharmacology, Karolinska Institute
  • Sophie Erhardt Dept. of Physiology and Pharmacology, Karolinska Institute
  • Maximilian Tufvesson Alm Dept. of Physiology and Pharmacology, Karolinska Institute
  • Burkhard Poeggeler University Hospital Schleswig-Holdstein (UKSH), Luebeck
  • Robert Schwarcz Maryland Psychiatric Research Center, University of Maryland

Keywords:

indole-3-propionic acid, kynurenic acid, neuroprotection, microbiota, tryptophan

Published

2024-04-23

How to Cite

Blanco Ayala, T., Sathyasaikumar, K., Zheng, Y., Schwieler, L., Erhardt, S., Tufvesson Alm, M., Poeggeler, B., & Schwarcz, R. (2024). The bacterial metabolite indole-3-propionic acid (IpRA) increases brain levels of kynurenic acid (KYNA) in an in vivo murine model. Archivos De Neurociencias, 29(S1). Retrieved from https://archivosdeneurociencias.org/index.php/ADN/article/view/607